sodium channel การใช้

• SKF-83, 959 inhibits sodium channels as well as delayed rectifier potassium channels.
• Unlike CFTR chloride channels, sodium channels behave perfectly normally in cystic fibrosis.
• On mammalian sodium channels, the type I toxins target receptor site 3.
• Furthermore, AM404 inhibits sodium channels, as do the anesthetics lidocaine and procaine.
• Sodium channel blocker lists both extracellular and intracellular classes of these substances.
• There is also an inward sodium current carried by cGMP-gated sodium channels.
• Aldosterone activates sodium channels that result in sodium resorption from the urine.
• Bifenthrin will open the sodium channel for a shorter period than other pyrethroids.
• The effect on sodium channels could be of particular relevance for seizure protection.
• Aconitine is a potent neurotoxin that opens tetrodotoxin sensitive sodium channels.
• Phenytoin binds preferentially to the inactive form of the sodium channel.
• It functions by opening sodium channels in the insect's nerve cells.
• Menthol also blocks voltage-sensitive sodium channels, reducing neural activity that may stimulate muscles.
• Sodium channels are known to be less selective in comparison to potassium channels.
• A remotely related group of atracotoxins operate by opening sodium channels.
• The site of action of TTX on the sodium channel has been identified.
• Sodium channels have similar functional properties across many different cell types.
• Voltage-sensitive sodium channels become persistently active at the resting membrane potential.
• Several electrophysiological studies have investigated the effects of lamotrigine on voltage-dependent sodium channels.
• The ?3 subunit can increase persistent current in certain sodium channels.